The Blow Lab:

CR-UK Chromosome Replication Research Group

Alberto Moreno

Postdoc

Tel: (+44)-1382-385833

Fax: (+44)-1382-388072

e-mail: a.moreno@dundee.ac.uk

Moreno A, Soleto I, García-Sanz P, Moreno-Bueno G, Palmero I (2013). ING4 regulates a secretory phenotype in primary fibroblasts with dual effects on cell proliferation and tumor growth. Oncogene, in press.

Culurgioni, S, Muñoz, I, Moreno, A, Palacios, A, Villate, M, Palmero, I, Montoya, G, and Blanco, F. (2012). The crystal structure of the inhibitor of growth 4 (ING4) dimerizationdomain reveals the functional organization of the ING family of chromatin binding proteins. J Biol Chem 287, 10876-84.

Abad, M, Moreno, A, Palacios, A, Narita, M, Blanco, F, Moreno-Bueno, G, Narita, M, and Palmero, I. (2011). The tumor suppressor ING1 contributes to epigenetic control of cellular senescence. Aging Cell 10, 158-71

Moreno, A, Palacios, A, Orgaz, J, Jimenez, B, Blanco, F, and Palmero, I. (2010). Functional impact of cancer-associated mutations in the tumour suppressor protein ING4. Carcinogenesis, 31, 1932-8

Palacios, A, Moreno, A, Oliveira, B, Rivera, T, Prieto, J, García, P, Bernadó, P, Palmero, I, and Blanco, F. (2010). The dimeric structure and the bivalent recognition of H3K4me3 by the tumor suppressor ING4 suggest a mechanism for enhanced targeting of HBO1 complex to chromatin. J Mol Biol 396, 1117-27

Gómez Cabello, D, Callejas, S, Benguria, A, Moreno, A, Alonso, J, and Palmero, I. (2010). Regulation of the microRNA processor DGCR8 by the tumor suppressor ING1. Cancer Res 70, 1866-74

Menéndez, C, Abad, M, Gómez-Cabello, D, Moreno, A, and Palmero I. (2009). ING proteins in cellular senescence. Curr Drug Targets 10, 406-17

Lachaud, C., Moreno, A., Marchesi, F., Toth, R., Blow, J.J. and Rouse, J. (2016). Ubiquitinated Fancd2 recruits Fan1 to stalled replication forks to prevent genome instability. Science 351, 846-849.

Moreno, A., Carrington, J.T.,  Albergante, L., Al Mamum, M., Haagensen, E.J., Komseli, E.-S., Gourgolis, V.G., Newman, T.J. and Blow, J.J. (2016). Unreplicated DNA remaining from unperturbed S phases passes through mitosis for resolution in daughter cells. Proc Natl Acad Sci USA, in press.